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Muscular AAS users engaged in heavy weightlifting can display rhabdomyolysis (307), sometimes with massive elevations of serum creatine kinase levels (308,–311), leading to myoglobinemia, myoglobinuria, elevated creatinine levels, decreased glomerular filtration rate (312), and the occasional progression to acute renal failure (51, 308). Notably, one recent case series has documented 10 cases of focal segmental glomerulonephritis among frequent AAS users (313), does hgh fix back problems. AASs may occasionally cause hepatotoxicity, with consequences including peliosis hepatis (an accumulation of blood-filled cysts in the liver) (314,–316), and various types of hepatic tumors (316,–322). Virtually all AAS-associated hepatotoxic effects are associated with orally active 17α-alkylated AASs (321, 323,–325). The frequency of AAS-induced hepatotoxicity is likely overestimated, however, because rhabdomyolysis from heavy workouts can increase transaminases (307, 326), and this finding may be erroneously interpreted as evidence of abnormal liver function (327).
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